Step 1: The key features are very low total protein and albumin (hypoalbuminaemia) with a distended abdomen (oedema/ascites) and NO proteinuria, in a young child with anaemia.
Step 2: Low albumin without protein loss in the urine points to inadequate protein intake rather than nephrotic syndrome. The combination of hypoalbuminaemia plus oedema (distended belly) in a toddler is the classic picture of kwashiorkor, a form of severe acute malnutrition.
Step 3: Kwashiorkor features oedema, hypoalbuminaemia, a fatty enlarged liver (adding to the distension), skin and hair changes, and apathy, distinguishing it from marasmus.
Why not the others: Marasmus shows severe wasting WITHOUT oedema and usually with relatively preserved albumin. Indian childhood cirrhosis causes hepatomegaly and liver failure but not this hypoalbuminaemic oedematous-malnutrition picture. Absence of proteinuria rules out a renal (nephrotic) cause.
Ref: Nelson Textbook of Pediatrics, 20th edition, Pg 301.